Yeah, I'll wait for that power to come back on.
Age old, as in the last 40 or so years, or how old exactly? And tested by whom?
I'll wait for that power to come back on before responding to this.
Don't you mean LCHP (Low Carb High Protein)? Could you perhaps be more specific here? State in a little more detail which foods "they" are eating causing them to over indulge on trans fats?
...waiting for the power to come back on.
Which "bad health risks"? Waiting for that power to come back on...
Agreed. That's why LCHF as espoused by Tim Noakes et al steers you away from trans fats.
Actually, it shows multiple health benefits: blood sugar level control, weight control and, iirc, reduction in the small dense LDL cholesterol particles in the blood (which contributes to heart disease).
But we'll wait for the power to come back on, and have a look at these proper, conclusive studies you know of.
You really don't want to learn do you? Replying with such confidence making NO sense what so ever.
Let me explain in detail then:
If you have high levels of cholesterol (which you probably do if you’re eating a high-fat, low-carb diet, then you need to be worried if your HS-CRP levels (a primary marker of inflammation) are above 1.0 mg/dL – even if you’re a hard charging athlete. I like to see most people under 0.5 for CRP levels, and here’s why: a high amount of inflammation in your body is going to make the cholesterol circulating in your bloodstream more likely to become oxidized, generating a high amount of heart and connective tissue-damaging free radicals. As a matter of fact, it’s more dangerous to have high levels of cholesterol and high levels of CRP than low levels of cholesterol and high levels of CRP – even if your high levels of cholesterol are “healthy”, big fluffy LDL particles, and not small, dense vLDL particles.
Free-ranging glucose molecules in your bloodstream can adhere to cholesterol particles and cause those particles to remain in the bloodstream for long periods of time, since your liver can’t properly process cholesterol when it has a glucose molecule attached to it. The longer cholesterol circulates in your bloodstream, the higher the likelihood that it will dig its way into an endothelial wall and potentially contribute to atherosclerosis or plaque formation. This is why it’s so dangerous to eat a high-fat diet, but to also have your nightly dark chocolate bar, overdo it on the red wine, or have weekly “cheat days” with pizza, pasta, or sugar-laden ice cream. If you’re going to eat a high fat diet, then you need to ensure your fasted blood glucose levels are staying at around 70-90mg/dL, and your hemoglobin A1C (a 3 month “snapshot” of your glucose) is staying below 5.5. If not, your high fat diet could actually be significantly hurting you.
Carbohydrates are necessary for the conversion of inactive thyroid hormone to active thyroid hormone, and if you’re on an extremely strict low carbohydrate diet, then you may actually be limiting this conversion. Your TSH is what tells your thyroid gland to “release more hormone,” so your TSH rises when your thyroid gland is underactive, or conversion of inactive to active thyroid hormone is inadequate. A high TSH means that the pituitary gland is releasing its hormone to try to get the thyroid to respond and produce more thyroid hormone. Because of inadequate carbohydrates, TSH will often elevate in a high-fat, low-carber – indicating potential for long-term thyroid and metabolic damage. If I see a TSH above 2.0 or a trend towards higher values in someone who is testing repeatedly, I get worried – and prefer to see TSH at 0.5-2.0. Of course, this doesn’t mean that you begin to shove carbohydrates indiscriminately down the hatch. However, it means that your high-fat, low-carb diet should include thyroid supporting foods rich in iodine and selenium, such as sea vegetables and brazil nuts, and should also include carbohydrates timed properly, such as before, during or after workouts, when the carbohydrate is more likely to be utilized for energy and less likely to spike blood glucose levels.
Lets no go onto you and Mr Noakes, now see everyone on this forum knows I fcking hate that guy NOAKES = DEVIL, I don't like him. Now he is a guy who makes huge statements without backing it up.
Noakes also argued that the conventional wisdom associating high fat consumption with heart disease was “unsubstantiated dogma”. (He has subsequently declared that carbohydrates are also responsible for all cancers. “Cancer is a carb-dependent disease”, he stated on Twitter.)
Noakes’ statements in relation to heart disease were bought to the attention of South African-trained Dr Jacques Rossouw, who works in the Cardiovascular Science division of the American National Heart, Lung, and Blood Institute (NHLBI).
At the end of 2012, on a family visit to Cape Town, Rossouw was invited to take part in a debate with Noakes at UCT – on the topic of cholesterol as a risk factor for heart disease, and current dietary recommendations.
The debate didn’t go well for Noakes. Facing an audience made up largely of academics and health professionals, his theories were aggressively dismissed. Noakes later called it a “kangaroo court”, and reportedly suggested Rossouw had been flown out “by the Heart Foundation ‘and other bodies’ to silence him”.
While much of Rossouw’s presentation (which is available for download from UCT’s health faculty website) revolved around complicated technical discussions on cholesterol and coronary heart disease (CHD), the correlations Rossouw drew between diet, obesity and chronic ill-health painted a slightly different picture to that posited by Noakes.
Rossouw’s data clearly indicated that, “reduced intakes of total fat, saturated fat and cholesterol, and increases in unsaturated fats were associated with reductions in serum cholesterol and [reduced] risk of CHD”. In other words: eating less fat had a positive effect on heart health.
Rossouw’s data showed something else: in certain populations where low-fat, high-carb diets were introduced, this had little to no effect on overall obesity. In countries like the US, Rossouw noted low-fat, high-carbohydrate eating practices had long preceded the current obesity epidemic – by several decades – indicating “other factors were primarily responsible”.
Importantly, even as obesity kept rising, CHD rates continued to decline where populations had reduced intake of dietary fat.
Rossouw went even further and suggested that, no matter what combination of fat versus carb we ate, obesity rates in developed countries would continue to rise. Changing dietary guidelines in Sweden provided proof. For around 30 years (from the 1970s onward), Sweden recommended and practised a low-fat “modified Mediterranean diet” to address high rates of cardiovascular disease. In the early 2000s, Sweden suddenly switched to a high-fat, low-carb eating model. Under both regimens, obesity increased.
Something else happened, too. As fat intake increased so did Sweden’s serum cholesterol levels. “Swedish researchers do not yet know if this change reversed the previously declining CHD rates,” Rossouw told Men’s Health. “It will be interesting to watch this natural experiment. I would predict their CHD rates will start increasing.”
Noakes has since developed diabetes.
