On an ECG trace there is a ventricle pulse making the wave QRS and then the small T wave that resets the ions called repolarization as the heart preps itself for the next sequence.That's rough, sorry hear! Yeah, the sudden ones are still the worst. Prior to having mine I didn't really know all that much about HAs. Over the last couple of years since they happened I've done insane research - to try and extend my own life, as well as give me some peace of mind.
My type was similar to your grandfather's: there would've been warning signs possibly prior to them happening. The usual stuff like slowing down physically, feeling over-exerted while doing basic things, a sharp pain (angina) when climbing stairs, etc. They're all trying to tell you the heart muscle is suffocating. Then there's the other kind - the type that killed both my parents. Sudden. No warning signs. And instant death basically:
My dad died when I was 16. He was in the workshop at work one morning, and then suddenly just collapsed. He was 47. My mom had fallen asleep on the couch while watching TV on Christmas day. She shot up clutching her chest, and that was it. She was 64. They were both relatively "normal" and had never displayed the symptoms I did, that I'm aware of. Sure, they smoked and weren't super active. But no prior issues really.
It's also the type of HA the drops someone running a marathon, so there's no real way to avoid it, I don't think.
The duration from Q to the end of the T wave must not be longer than 470ms.
If it is longer due to genetics or medication then it is called Long QT SYNDROME (LQTS). This can precipitate to Torsades de Pointes (TdP) and unconscious and death if it deteriorates to Ventricular Fibrillation (VT).
Many athletes, soccer and rugby players have had a sudden death on the field due to this. Cot deaths are also thought to be caused by TdP
If no cause for sudden death can be found then it is highly likely to be due to LQTS.
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